Nuclear event.

Any student of radiobiology knows our understanding of radiation-induced carcinogenesis is heavy in theory and light in data. Here’s an in-depth genomic look at 359 papillary thyroid tumors among survivors of the 1986 Chernobyl accident with an additional cohort of 81 cases among those born after 1986. Driver aberrations were identified in 98% of cases, the majority of which were in the MAPK pathway and often BRAF mutations or RET fusions. Radiation exposure was quantitatively estimated based on patient location at the time of the Chernobyl accident and, indeed, there was a linear dose response for radiation-associated molecular characteristics such as nonhomologous end joining repair of double-strand DNA breaks that ultimately resulted in pathologic fusions. | Morton, Science 2021

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